The brain encompasses numerous kinds of cells, such as neurons, astrocytes, microglia, oligodendrocytes, pericytes, and endothelial cells, each with specific structures and functions that play an integrated role in CNS homeostasis. For instance, glial cells—especially astrocytes and microglia—respond to immunological signals, secrete chemokines, cytokines, and growth factors, perform phagocytosis, and provide essential support to neurons and the vasculature of the CNS.
Any insult to the brain, such as a traumatic brain injury, triggers neuroinflammation, which can lead to chronic traumatic encephalopathy (CTE) and other neurodegenerative diseases. Environmental and occupational factors, such as exposure to blasts, can also spark neuroinflammation and cognitive impairment. Additionally, addiction to substances like alcohol and tobacco, as well as hormonal imbalances, can significantly impact brain health.
Furthermore, the health of other organs, including oral health and microbiota, has profound implications for neurological and cardiovascular well-being, a topic that has garnered significant attention in recent research. With rapid advances in neuroimaging, improved diagnostic tools, and the identification of biomarkers, our understanding of brain pathologies and their chronic effects has evolved dramatically.
This Special Issue is the second installment in a captivating series dedicated to these critical topics. It features a collection of intriguing research articles on cellular functions, environmental impacts, and therapeutic interventions for brain pathologies, providing a comprehensive overview of the current progress in neuroscience.
With a compilation of 16 insightful articles, this Special Issue promises to be a fascinating read for researchers, healthcare professionals, and anyone interested in the complexities of brain health. Whether you are a student of neuroscience or a practitioner seeking the latest insights, this collection is an essential
Authors:
Kumar Vaibhav (Editor)
Edition:
Publication Date:
March 7, 2025
From the book:
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